Neurogenesis and neuroplasticity mechanisms
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Latest in Neurogenesis and neuroplasticity mechanisms
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A transcriptional switch from KLF activators to repressors during early postnatal development regulates neuronal maturation in the mouse neocortex.
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Early lineage divergence in the embryonic mouse thalamus segregates sensory and motor-associative identities from distinct progenitor domains.
- Open access
Gαi2 promotes microtubule-driven neurofilament incorporation to stabilize nascent axons in the developing chick spinal cord.
- Open access
Microtubule pushing forces are essential for preventing spindle stalling, enhancing robustness, and facilitating alignment alongside cortical pulling in diverse cell types.
- Open access
TDP2-mediated DNA repair is essential for timely regulation of early response gene transcription in cortical neurons, with Tdp2 deletion causing ERG overexpression and circuit dysfunction.
- Open access
Reactive astrocyte-like tumor cells with mesenchymal and injury-response signatures are enriched after radiotherapy and persist in recurrent glioblastoma in a mouse model.
- Open access
Human peripheral-central dual neural organoids (hPCOs) reveal bidirectional Wnt signaling roles in neurodevelopment, modeling nociceptor defects and teratogenic risks.
Proprioceptive sensory neurons are essential for the postnatal development and organization of Clarke's column neurons in the spinal cord.
- Open access
Myeloid-specific Tet2 deletion generates myeloid-derived activating cells that enhance radiotherapy and suppress metastasis in lung cancer models.
Human-specific morphoregulatory enhancer activity and gene expression signatures distinguish basal radial glia from other great apes, contributing to neocortex evolution.
- Open access
Reelin deficiency causes persistent disruptions in primary cilia orientation, localization, and length in principal neurons of the cerebral cortex, especially in laminated regions.
- Open access
- 1 cites
Gata4-Loxl2b axis is essential for ECM remodeling and scar-free spinal cord regeneration in zebrafish, with loss of Gata4 impairing recovery.
- Open access
Oligodendrocytes actively support CNS function through metabolic provision, adaptive myelination, and repair, but become dysfunctional in aging and disease.
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