Biofilms enable bacteria to resist antibiotics through complex molecular networks, with over 70% of persistent infections linked to biofilm formation.
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Moving in Virulence.
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Biofilms enable bacteria to resist antibiotics through complex molecular networks, with over 70% of persistent infections linked to biofilm formation.
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Proteomic profiling reveals that the (p)ppGpp-mediated stringent response in Pseudomonas aeruginosa regulates over 339 proteins during stationary phase, highlighting extensive metabolic shutdown and stress adaptation.
The bacteriophage transcription factor Gp53 suppresses Pseudomonas aeruginosa growth, motility, and biofilm formation by regulating key host genes.
Mechanical compression activates cAMP signaling in Pseudomonas aeruginosa with an estimated force of 3-30 nN, linking physical stimuli to virulence regulation.
Distinct lignocellulosic and cellulose-derived carrier classes are analyzed for biofilm-based food fermentations, emphasizing their structural, functional, and validation differences.
YdzF, a non-canonical regulator, controls bacterial nanotube formation in Gram-positive bacteria through YmdB-dependent flagellar gene regulation.
SMF-1 pilus forms antiparallel bundles that facilitate rapid biofilm initiation in Stenotrophomonas maltophilia, revealing a unique structural mechanism for bacterial colonization.
Synthetic biology approaches to quorum sensing can be harnessed to develop innovative antimicrobial strategies targeting bacterial virulence and resistance.
A rapid sphingomyelin-dependent entry pathway in S. aureus alters intracellular fate, influencing bacterial replication and host cell death within minutes of contact.
Sodium hydroxide induces 45% higher cell lysis in Bacillus coagulans during fermentation by causing energy deficits under carbon source transition.
Nearly half (46.7%) of street food samples in Bangladesh contained Klebsiella pneumoniae, with 37.5% showing multidrug resistance and high virulence gene prevalence.
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