Cardiac Fibrosis and Remodeling
Moving in bioRxiv, Frontiers in Genetics, medRxiv, Nature Communications, eLife, EMBO Reports and Nature Medicine.
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Latest in Cardiac Fibrosis and Remodeling
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Sulf1 modulates TGF-β-dependent epicardial activation and inflammation, with its disruption causing regeneration defects and increased fibrosis in zebrafish hearts.
- Open access
S1PR1, CA2, and ABCG2 are identified as potential diagnostic biomarkers for pulmonary arterial hypertension, with S1PR1 showing a protective role in endothelial cells.
- Open access
Ferroptosis-related transcriptomic patterns in dilated cardiomyopathy are donor- and context-dependent, with no evidence for ferroptosis as a causal mechanism in primary hypertensive heart disease.
- Open access
Primordial cardiomyocytes are essential for heart development but not necessary for regeneration in zebrafish, with only 15% capable of regeneration after injury.
- Open access
Mitiperstat, an MPO inhibitor, did not improve symptoms or exercise capacity in 711 patients with heart failure and preserved or mildly reduced ejection fraction over 48 weeks.
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