Type I interferon promotes ILC2-to-ILC1 plasticity, contributing to radiation-induced lung injury in a mouse model.
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Type I interferon promotes ILC2-to-ILC1 plasticity, contributing to radiation-induced lung injury in a mouse model.
Glutamate suppresses ILC2 responses and reduces allergic airway inflammation via the kainate receptor GluK2 in mice.
The H3K27me3-NR1D1-IFN-γ axis in lung-trained macrophages associated with early respiratory viral infection drives non-T2 asthma in childhood mice.
BHLHE40 orchestrates intestinal immunity by regulating ILC3s and RORγt⁺ APCs, crucial for mucosal defense and tolerance, with a 40-fold increase in expression during inflammation.
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