Multiscale modeling shows that endothelial repair under shear stress depends on nonlinear AKT/NO/ROS interactions, with a critical intervention window on Day 1.
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Moving in bioRxiv, eLife, Nature Plants and PLOS Computational Biology.
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Multiscale modeling shows that endothelial repair under shear stress depends on nonlinear AKT/NO/ROS interactions, with a critical intervention window on Day 1.
SUMO2 modification of p66Shc at lysine-81 mediates hyperlipidemia-induced endothelial dysfunction in mice.
A leucine-rich repeat receptor kinase called CERI is essential for systemic nitrogen-demand signalling in plants, with CERI loss impairing this process in Arabidopsis.
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Topics moving, week to 23 Oct 2026